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Three possible forms of interaction involve the reaction of o-quinone with sulfhydryl residues of cysteine or with amino groups of lysines, and a third possibility is the reduction of the semiquinone radical by a sulfhydryl group [170] (Figure 12)
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This could account for the selectivity of the pro-apoptotic effect of CBD in activated HSCs, wherein CBD exacerbates the pre-existing ER stress response, thereby switching signaling from adaptive to apoptotic
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