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The alteration of H3K36me3 was found to be higher in the promoter regions of oxidative stress response (OSR) genes in HaCaT and HEK cells [103]
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Although many pro-tumorigenic effects of IGF axis activation are mediated via IGF-1Rs expressed by target epithelial, mesenchymal, and endothelial cells, it is increasingly recognized that IGFs also influence anti-tumor immunity
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