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In this review, we primarily use the term LPD to refer to inadequate corpus luteum function characterized by suboptimal progesterone secretion or shortened progesterone exposure, which in turn compromises endometrial receptivity for implantation and early pregnancy maintenance [9, 10]
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In this assessment of current knowledge, we highlight the central position of the ascorbate–glutathione cycle in the network of cellular redox systems that underpin the energy-sensitive communication within the different cellular compartments and integrate plant signalling pathways
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